Mice with hyperbilirubinemia due to Gilbert’s syndrome polymorphism are resistant to hepatic steatosis by decreased serine 73 phosphorylation of PPARα

Keyphrases

• Bilirubin 10%
• Blood Glucose 10%
• Blood Insulin 10%
• C57BL 10%
• Conjugation 10%
• Fasting Plasma Glucose 10%
• Fat Burn 10%
• Fat Mass 20%
• Gilbert 20%
• Gilbert Syndrome 100%
• Hepatic Lipid 30%
• Hepatic Lipid Accumulation 10%
• Hepatic Steatosis 100%
• High-fat Diet 10%
• Humanized Mice 20%
• Hyperbilirubinemia 100%
• Immunostaining 20%
• Insulin Level 10%
• Lean Body Mass 10%
• Lipid Content 20%
• Liver 10%
• Liver Sections 10%
• Low Serum 10%
• Peroxisome Proliferator-activated Receptor 100%
• Phosphorylation 100%
• Polymorphism 100%
• Protein Level 10%
• Receptor Activation 10%
• Regulated Genes 10%
• Serum Insulin Levels 10%
• Total Bilirubin 10%
• Total Body Lean Mass 10%
• Transcriptional Activity 30%
• UGT1A1 10%
• Unconjugated Bilirubin 10%

Biochemistry, Genetics and Molecular Biology

• Bilirubin 20%
• Bilirubin Blood Level 10%
• Body Weight 10%
• Fat Mass 10%
• Glucose Blood Level 20%
• Glucose Level 10%
• Immunostaining 20%
• Insulin Blood Level 20%
• Insulin Level 10%
• Lean Body Weight 10%
• Lipid Liver Level 30%
• Lipid Storage 10%
• Mouse 100%
• Peroxisome Proliferator-Activated Receptor 100%
• Serine 100%